Recurrent coronary insufficiency was simulated in animals using histochemical and histological techniques, and electron microscopy. Studies of the myocardium at recurrent coronary insufficiency of blood supply enabled us to reveal substantial changes in all structural and functional links responsible for maintenance of vital functions of cardiomyocytes (cardiac hystiocytes). These changes inevitably resulted in redistribution of the functional load between the altered cardiomyocytes; some of them functioned under conditions of a chronic energy deficiency, electron misbalance, and disrupted regulation of plastic processes. This emergent heterogeneity of the myocardium at each coronary crisis is a qualitatively new event that substantially re-build the morphofunctional properties of the myocardium, and probably also its reactivity.

  Якщо, ви не знайшли інформацію про автора(ів) публікації, маєте бажання виправити або відобразити більш докладну інформацію про науковців України запрошуємо заповнити "Анкету науковця"